Tag Archives: Plagiarism

Intriguing finding in study of neglected children suggests another one of my (possibly) novel neurodevelopmental hypotheses

Do some neglected or sound-perception-impaired children teach themselves how to amuse themselves by simply looking at and silently analysing their surroundings, and thus develop an inferior temporal lobe that is more developed than it would otherwise have been within the context of brain-stunting deprivation, and in doing this, do these kids gain an advantage over other neglected kids (who will develop ADHD-type behaviours) in learning how to focus their attention and control their own behaviour?* Could this hypothesis help us to understand the development of conditions and abilities associated with strengths and unusual activity in visual processing, things such as hyperphantasia, autism, superrecognition or forms of synaesthesia that involve visual inducers or concurrents (which is just about all of the recognised forms of synesthesia)?*

Seems a bit controversial that this radio story has linked disorders such as autism and ADHD with childhood neglect, but this also sounds very plausible to me, keeping in mind that some kind of unidentified and unknown perceptual disorder in a child or infant could cut the child off from their environment in a way that would mimic extreme childhood neglect, so evil parents are not necessarily a part of a hypothesis based in this idea. I think this is all there is to “autism” – some perceptual (not sensory) disability stopping normal development in communication abilities that the world’s autism experts have not identified or researched.* “Autism” is such a massive cash-cow for so many people in respected positions, it would really upset the apple-cart if its causal mechanism was identified and a remedy found.

*Don’t forget – don’t plagiarise my ideas.

Romania’s orphans — early neglect, brain size and behaviour
Health Report
ABC Radio National

Guest: Professor Edmund Sonuga-Barke   Professor of Developmental Psychology, Psychiatry and Neuroscience, King’s College London

Host: Dr Norman Swan

Producer: James Bullen

Broadcast: Mon 27 Jan 2020.

https://www.abc.net.au/radionational/programs/healthreport/neglected-children-have-smaller-brains/11893144

 

Looks like I’m not the only librarian

who has been screwed-over by Frontiers:

http://www.nature.com/news/controversial-website-that-lists-predatory-publishers-shuts-down-1.21328

Beall-listed Frontiers empire strikes back

http://www.nature.com/news/backlash-after-frontiers-journals-added-to-list-of-questionable-publishers-1.18639

 

Amazed, not in a good way

The 2013 journal paper that ripped-off my excellent and original idea linking synaesthesia with specific elements of the immune system (published in this blog in 2012) has been reprinted as a chapter in a book that was published just a few months ago. One of the book’s authors is also one of the two authors of that paper, and another one of the book’s authors was an editor of that paper. Have these people no shame?

https://books.google.com.au/books?id=Tmy5CgAAQBAJ&source=gbs_navlinks_s

The offending paper has also been cited in a 2015 paper by two of the book’s authors

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4403345/

Also not nice to know that recent work of those researchers was scheduled to be presented by one of them at the Eleventh Annual National Conference of the American Synesthesia Association at the University of Miami, Florida, held last month. This year’s conference was organized by the same academic who was the reviewer of the offending 2013 journal paper.

http://www.synesthesia.info/upcoming.html

http://www.as.miami.edu/media/college-of-arts-and-sciences/content-assets/philosophy/docs/October%202-4%202015_Synesthesia_Program-miamiflorida.pdf

And one of those researchers will be the supervisor of a PhD studentship beginning January 2016

http://www.sussex.ac.uk/study/money/scholarships/pgr2016/view/541

I’d like to invite those involved (so many of them) to individually or collectively go and dip their left eyes in hot cocky cack, to quote an Australian cinema classic

http://aso.gov.au/titles/features/adventures-barry-mckenzie/clip1/?nojs=Ver

 

 

All those years of neuroimaging research on the brains of synaesthetes has found nothing of substance?

Hupé J and Dojat M (2015) A critical review of the neuroimaging literature on synesthesia. Frontiers in Human Neuroscience. 9:103.
doi:10.3389/fnhum.2015.00103

http://journal.frontiersin.org/article/10.3389/fnhum.2015.00103/abstract

“Our critical review therefore casts some doubts on whether any neural correlate of the synesthetic experience has been established yet”

That is a bit of a shock to read. This isn’t the first time that I’ve gotten a big shock after reading a paper in the journal Frontiers in Human Neuroscience. There was that little matter of some of my most amazing neuroscientific ideas published at this blog being ripped-off and used as the guts of an “opinion article” in that journal in 2013. I haven’t forgotten that episode. Who would have thought so much excitement is there to be found inside a science journal? I should make it clear that the researchers who did that thing in 2013 are NOT the authors of the above paper, but at the same time, I’ve got to wonder where Hupé and Dojat got this idea from

“…synesthesia could be reconsidered as a special kind of childhood memory, …”

Sure, they could have thought of that under their own steam, but I still want to point out that the central, seminal idea of this blog, right from the very first post in 2010, has been the idea that synaesthesia is linked in some meaningful way with face memory, in my case with super-recognizer ability in face memory, and there are many articles in this blog that show and hint that the heart of synaesthesia is memories created in childhood and many different types of synaesthesia operate in ways that are so much like memory that the differences are only quantitative. There was even one article published in 2013 at this blog in which I stated that

“…the Proust phenomenon is considered to be a type of memory and many of my observations at this blog have demonstrated that synaesthesia can involve memory, is an element of the “method of loci” memory technique and I would argue operates like memory. Yes, Yes, Yes, the Proust Phenomenon is a close relative of synaesthesia.”

Some ideas that I’d like to (explicitly) lay claim to (right now) in 2014

Would you like to follow these steps?

First go the the Internet Archive (Wayback Machine):  https://archive.org/

Then copy and paste the web address of this blog into the search form that you will find there, removing the beginning bit, and click on “BROWSE HISTORY”:   https://superrecognizer.wordpress.com/

The results screen that comes up will show you how many times this blog has been archived by the Internet Archive over the years.

Click on the year 2012, then click on the date June 21st 2012.

The Internet Archive will display to you their archived record of the home page of this blog as recorded on June 21st 2012. You will see a blog coloured in green and blue with stories featuring photos of some sculptures seen around Perth, but the thing that you might (or might not) find interesting is the blog post that is third down the page which was first published on June 7th 2012. This is the blog post where I briefly but clearly published my ideas suggesting causal relationships between the human immune system and synaesthesia, and low levels of some of the complement immune chemicals and synaesthesia, all related to the regulation of developmental synaptic pruning and synaptic plasticity involving the activity of microglia, and in this post I also restated my previously-published speculation that the variety of dementia known as Benson’s syndrome or posterior cortical atrophy could be regarded as the opposite of developmental synaesthesia associated with special abilities in visual perception such as super-recognition ability and exceptional reading ability, a cluster of traits that appears to run in at least one family. Got that? The point I’m trying to make is that I published this stuff in June 2012, I thought of these ideas independently and as far as I can tell no one else had previously published these ideas anywhere, including in scientific journals.

Now take a look at this paper in a neuroscience journal which was published in 2013 and was received by the journal as a draft or manuscript on July 31st 2013:  http://journal.frontiersin.org/Journal/10.3389/fnhum.2013.00563/full

Do any of the themes in the paper seem familiar? Does my blog or myself receive any credit or acknowledgement in the paper? Hmm.

Regardless of any issues related to originality or acknowledgement, the important point in all of this is that here we have some ideas about a type of dementia which could conceivably have some medical or scientific use or value. My idea of linking synaesthesia with the immune system is nice but just a step in a possibly much more important sequence of ideas. I’d like to give those ideas another airing, while also restating that I thought of them a long time ago independently and claim all due credit. I hope you don’t mind.

Thank you PubPeer

Thank you PubPeer for (after some fussing about) giving me a place on the internet that is recognized by scientists to publish my objection to some researchers publishing a highly original idea that I had thought of independently and had published at this blog over a year before their draft paper was received by their publisher. I’m certain that this is a case of plagiarism, but other people object to my use of that word, so the only place you will see the word plagiarism used in relation to that matter is here at this blog.

I have already published a very full account of my side of the story at this blog, but unfortunately this blog isn’t recognized as a part of the ecosystem of commentators attached to the world of science, the people and organizations in the league of science journalists, science magazine bloggers and internet services that are supposed to review the science literature but actually appear to be automatically-generated content. PubPeer isn’t like that; it appears to be run by people, but who they are is a mystery. I can completely understand why they wish to remain anonymous. Exposing the many ways in which peer review in science publishing is broken is a pastime that I am sure could be harmful to one’s career in science.

I believe that I can make a greater contribution to science as a blogger who has no job that is in any way connected to any university or any research institution, because I don’t have to deal with career-building and politics that goes with having that kind of career, and as a result I have more “mental bandwidth” free to devote to thinking about actual science. I am not expected to teach half-interested university students or organize conferences or write full-sized published papers or book chapters, and I’m not expected to know my proper place in the scheme of things. Very ordinary physical activities that I do in my everyday domesticated life automatically activate regions of my brain that deal with conceptual thinking and memory, and as a result I am often bombarded by novel ideas resulting from a boundless miscellany of concepts flashing onto the centre stage of my mind and colliding in a quite haphazard manner. I guarantee this kind of involuntary mental activity wouldn’t happen if I spent my days comfortably sedentary inside an airless office, staring at a wall or a dusty old painting. I’ve had jobs like that in the past, and years of study at an austere and ugly university which was also pretty much the staring at a wall lifestyle. I find it ironic that study at a university can offer an environment that has an effect on thought that must surely compare with a lobotomy.

Being a nobody to the world of science has many up-sides. I don’t feel a lot of need to consciously or unconsciously self-censor my ideas and publications to fit in with the beliefs and fashions of researcher peers (whether they make sense or not), and I don’t limit my thoughts to my job description, because I have no job description (and I also unfortunately have no salary, pay or financial benefit of any kind). Having no identity in the world of science means I also have no specialization or niche, leaving me free to see and write about a completely obvious connection between synaesthesia, an area of science typically researched by research psychologists and non-clinical neuroscience/psychiatry researchers, and the human immune system, an area of science that has I guess typically been researched by practicing medical doctors in the specialties of immunology or rheumatology. For sure there have been in the last ten years or so a group of pioneering and original researchers who have researched the incredibly complex ways in which the human immune system impacts on brain development, but I don’t think any of them have linked their work with synaesthesia or any particular type of dementia, as I have.  I’m guessing that they haven’t written about synaesthesia because they feel that it is too trivial a matter for them to bother with, which is probably a defensible point of view, as synaesthesia isn’t a disorder and I’ve never heard of a synaesthete who is looking for a cure. I feel free to write about concepts such as dementia, the complement immune chemicals and synaesthesia that are areas that are beyond my expertise, because I have no recognized area of scientific expertise or any recognized career in science. A lot of words have been written about the concept of intellectual freedom in academia, but I have found that being a non-entity outside of all that is a state that offers the most intellectual freedom. It’s a bit like being the invisible man; you can get up to all kinds of stuff but you can’t hope for recognition, because you have no face and no identity.

Being nobody is a state of freedom but it certainly has it’s frustrations, like having no income as reward for any of the work I put into this blog and the ideas published in it, and having no recognition, not even on the internet, but the most frustrating thing is the way that my most important idea has had no apparent impact in science. I’m not referring to my idea linking the immune system with synaesthesia, which was just an amusing step in my journey to the much more important idea that one particular type of dementia, known by the names Benson’s syndrome or posterior cortical atrophy or PCA, could be caused by an excess of one or more of the complement immune chemicals. Surely this is an idea that could be researched. Surely this is an idea that could lead to a number of different ideas for therapies if it turns out to have some value. Surely this is an idea worth at least checking, for if it reflects reality it surely has the potential to save minds and brains and lives. HELLO! Is anybody listening?

P.S. Late last month an Australian biotechnology company plunged on the stock market following the announcement of the failure of their drug aimed at treating Alzheimer’s dementia during a stage of a study. After years of hoopla and hype about drug companies finding a cure for Alzheimer’s or dementia, and anyone’s guess how much money spent on studies, all they can offer is symptomatic treatments. The story has been one failure after another. Benson’s syndrome is considered by some to be a variant of Alzheimer’s disease. Whether it is or not, maybe it is high time for researchers to stop obsessing about plaques in the brain and look at the immune system and the brain. My money is on C3 and C4 as concepts to focus on in a search for an understanding and cure for dementia. Would it kill you dementia researchers to accept some advice from a Perth blogger who is nobody in particular, and put my name on your research paper if my idea works out?

Upcoming Cortex special issues look interesting

http://www.journals.elsevier.com/cortex/forthcoming-special-issues/

I’ve got my eye and my occipital lobes and other well-connected parts of my brain focused on the upcoming Cortex special issues about visual cognition and neuroplasticity, because I have a particular interest in these quite exciting areas of research.

I do hope there will be no plagiarism or any other form of scientific mischief in these special issues. In October of last year Cortex published an editorial about plagiarism and how the editors are are committed to fight it, which is a bit of a coincidence considering that it was the same month in which I made allegations of plagiarism at this blog concerning a paper that had been published the month before by another neuroscience journal, and one of the authors of that paper works in the same university department as one of the co-authors of the Cortex editorial about plagiarism. Many times since October 2013 I have wondered about the nature of chatter around the water-cooler in that university department.

Some ideas that I’d like to (explicitly) lay claim to (right now) in 2014

A note of warning – If you are thinking about copying or plagiarizing any of the text, ideas or descriptions in this post or using it in your own work without giving me (C. Wright, author of the blog “Am I a Super-recognizer?”) the proper acknowledgement and citations, then think again. If you do that you will be found out and my objection will be well publicized. If you believe that you published any of these ideas before I did, please let me know the details in a comment on this article. If you want to make reference to this blog post or any of the ideas in it make sure that you state in your work exactly where you first read about these ideas. If you wish to quote any text from this post be sure to cite this post at this blog properly. There are many established citation methods. If you quote or make reference to material in this blog in your work, it would be a common courtesy to let me know about your work (I’m interested!) in a comment on any of the posts in this blog. Thank you.

The idea that Benson’s syndrome or posterior cortical atrophy or PCA, a variety of dementia, is caused or develops in a way that can be seen as the opposite of the synaesthesia linked with exceptional visual memory and literacy skills that runs in my family (this idea has been explored previously in this blog).

The idea that the above cited states develop or are caused in a way that makes them seem like opposites because they both affect the same or similar areas of the brain, but in opposite ways.

The idea that the above described process happens because Benson’s syndrome and our variety of synaesthesia are both mediated by the same or similar natural chemical or cells or biological agent in the brain, one caused by high levels of the mystery substance and the other caused by low levels (a hypothesis that I briefly suggested in January 2011).

The idea that one of the many known or unknown elements of the immune system that impact brain development is the mystery substance referred to above (a hypothesis that I briefly outlined in 2012).

The (implied in above ideas) idea of the immune hypothesis of synaesthesia. (This idea was first published by me in 2012 in a blog post archived here, was I believe plagiarized in 2013 here, and was the subject of my plagiarism claim here.)

The idea that one or more of the complement immune chemicals is the  mystery substance referred to above.

The idea that the C3 complement immune chemical  is the  mystery substance referred to above.

The idea that synaesthesia is linked with one or maybe more immune diseases or conditions caused by low levels of complement.

The idea that genes for synaesthesia stay quite common in the gene pool because of some associated cognitive advantage (probably superior memory) that balances out any disadvantages caused by deficiencies in the immune system.

The idea that some or many people unintentionally experience a memory process that operates in a similar way to the method of loci memory technique in their everyday lives, unintentionally forming long-term associations between individual learned concepts and individual visual memories of scenes (I have named this phenomenon Involuntary Method of Loci Memorization or IMLM).

The idea that IMLM operates in such a similar way to synaesthesia that one could argue that it is a type of synaesthesia.

The idea that synaesthetes are more likely to experience IMLM than non-synaesthetes.

The (implied) idea that the method of loci memory technique is similar to or a type of synaesthesia.

The idea that synaesthetes might have a natural advantage in using the method of loci because the method of loci is similar to or is a type of  synaesthesia. This idea that seems likely in light of the case of “S” the Russian memory performer with many types of synaesthesia described by Luria. 

The idea that IMLM is a phenomenon that is caused by enhanced synaptic plasticity throughout the life span.

The idea that IMLM is a phenomenon that is caused by enhanced synaptic plasticity throughout the life span and can thus be used as an indicator of which synaesthetes are synaesthetes due to enhanced synaptic plasticity throughout the life span rather than other possible causes of synaesthesia. Support for this idea comes from the fact that IMLM appears to be a non-developmental variety of synaesthesia that can form new long-term associations in adolescence and adulthood.

The idea that IMLM is a phenomenon that is caused by the unusual possession of levels of synaptic plasticity typical of a young child, during adolescence or adulthood.

The idea that IMLM is caused or enhanced by some characteristic of the immune system that affects the functioning of the brain. Many different elements of the incredibly complex immune system are thought to affect the functioning or development of the brain, and could thus be involved in IMLM, including the complement system, microglia and the MHC class I molecules. Researchers such as Beth Stevens and Carla Shatz have investigated this exciting area of neuroscience. In 2012 I hypothesized at this blog that synaesthesia could be caused by low levels of complement, this idea implying that the immune system is directly involved in synaesthesia (or at least some cases of synaesthesia). I believe these ideas were plagiarized in a paper published in 2013.

The idea that IMLM is similar to the “Proust phenomenon” in that it is very similar to synaesthesia or is a type of synaesthesia and involves episodic or autobiographical memory as a concurrent.

The idea that phonics as a foundational reading skill is similar to or is arguably a type of synaesthesia in that it involves the involuntary association of individual speech sounds with individual printed letters or combinations of letters, as the result of learning in early to mid childhood.

The idea that at least one type of dyslexia is like a deficiency of synaesthesia.

The implied idea that if synaesthesia has as it’s basis hyperconnectivity in the white matter of the brain, dyslexia as an opposite of synaesthesia or a deficiency of synaesthesia is or could be caused by hypoconnectivity in the white matter of the brain (I suspect there might be existing research evidence that supports this idea).

The implied idea that in at least one cluster or grouping of cases synaesthesia is associated with superiority in literacy or reading skill.

The idea that synaesthesia can happen in different regions of the brain, and because of this the experience of various types of synaesthesia can vary in detectable ways because of the influence on the synaesthesia of the varied ways that different areas of the brain operate. This can mean that one synaesthete can experience different types of synaesthesia that operate in very different ways, for example, some types of synaesthesia more rare or spontaneous or intrusive than other types. (I am not completely sure of the originality or the novelty of all of this idea.)

The idea that there is an association between synaesthesia and super-recognition that is not merely coincidental.

The idea that synaesthesia is a type of memory or learning. (Not sure if I’m the first to note this obvious fact).

The idea that synaesthesia concurrents are re-experienced memories, or re-activated “learnings” of concepts, not perceptions. (Not sure if I’m the first to note this obvious fact). In support of this idea I can assert that synaesthesia is like face recognition in that both are visual memory-based phenomena which are subject to the Verbal Overshadowing Effect or something very similar. My assertion that synaesthesia is subject to the verbal overshadowing effect is based on my own observations (outlined elsewhere in this post).

The idea that super-recognizers should or could be trained and employed as expert consultants in the practice of medical genetics.

The idea that medical geneticists and all types of medical specialists need to have a super-recognizer level of face memory or face recognition ability, so that they can intuitively and quickly recognize medical facies.

The idea that there is no clear point of distinction between medical facies or faces associated with genetic syndromes and normal faces.

The idea that super-recognizers could be used to facially identify blood relatives of a person or persons.

The idea that super-recognizers could be used to facially identify the specific ethnicity of a person.

(below ideas added January 28th 2014)

The idea that super-recognition or being a super-recognizer could develop as the result of an unusual level of fascination with the visual appearance of landscapes or scenes, rather than from a fascination with faces, and thus be a side-effect hyper-development of a part of the brain that serves two similar functions.

The idea that super-recognition or being a super-recognizer could, at least  in some cases, develop as the result of a general hyper-development of the visual sense to compensate for problems in the auditory sense during childhood such as temporary deafness, recurrent ear infections, glue ear or poor auditory processing.

(below idea added February 1st 2014)

The idea that lexical-gustatory synaesthesia is an exaggerated form of some kind of evolutionary adaptation in the brain that biologically primes the mind to attend to or react to speech on the subject of food (this idea was discussed at this blog in a post dated January 27th 2011, with more consideration in a later post).

(below ideas added February 6th 2014)

The idea that creativity might be immediately enhanced during and only during the duration of physical or visual-spatial activity because the activity activates areas of the brain associated with movement and in turn these areas activate other areas of the brain including those that give rise to conceptual thinking, and the increased activation makes novel associations between diverse thoughts and concepts more likely, and that this process is like synaesthesia or is a type of synaesthesia, and the types of physical activity that are the most effective inducers of this effect might be highly specific, highly specific in effects, highly varied between individuals and highly idiosyncratic, as is typical of synaesthesia inducers and concurrents. Driving a car can act as an inducer of this effect. (I have gone some way to exploring this idea in past posts.)

The idea that mental flexibility might be immediately enhanced by the above effect, which I will name “movement – thought-flexibility synaesthesia”.

The idea that thinking might be immediately enhanced by the above effect.

The idea that memory might be immediately enhanced by the above effect.

The idea that the above effect is similar to embodied cognition or is a type of embodied cognition.

(below ideas added February 14th  and  February 20th 2014)

The idea that synaesthesia is like the process of face recognition (and vice versa), because they both

– are subject to the verbal overshadowing effect or something similar

– are automatic

– are involuntary

– have a sensory inducer, in face recognition always visual, in synaesthesia I think most frequently visual

– have or can have a concurrent that could be described as a memory, a concept or a personality (I’m comparing face recognition with personification synaesthesias and the synaesthesias that I have described at this blog which have visual memories of scenes as concurrents)

– are or can be visual in both the inducer and concurrent

– typically involve the fusiform gyrus

– involve set pairings of inducers and concurrents (same person’s face seen before then recognized later)

– involve set parings of highly specific inducers and concurrents (I recognize that an employee at my local supermarket has a sister who has just started working there too, as their faces and bodies and hair are near-identical, but for the extra acne and the more receding chin of the new employee. They are very similar in appearance but my discrimination is highly specific, just as I can recognize that the green wall on the lower floor of a public library is close to but not quite the same colour as Tuesday.)

– both can have, but do not always have an actual face as an inducer (we can recognize the faces of celebrities in photos, caricatures and art, even seeing Marilyn Monroe’s face in a pattern of brown coffee cups stuck to the wall at the coffee shop at the art gallery.)

(below idea added February 17th 2014)

“My particular interest in personification is my own theory that personification synaesthesia (as experienced by myself) or something like it gives rise to superiority in face memory (or being a super-recognizer) by naturally making the faces of unknown people more memorable and interesting”

The above is a quote from an article that was published at the blog in October 2013.

(below ideas added February 19th 2014)

The idea that the synaesthesia brain is the result of the developmental influence or shaping from, or the adaptation to, the behavioural phenomenon of “flow” as described by Mihaly Csikszentmihalyi.

The idea that synaesthesia, intellectual giftedness or high IQ and autism or Asperger syndrome seem to coincide more often than chance because gifted and autistic kids are more likely to experience “flow” and this in turn can influence the developing brain in a way that gives rise to synaesthesia.

(below ideas added February 20th 2014)

The idea that the genuine conscious awareness of synaesthesia is a threshold phenomenon that operates in conflict or competition with conscious thinking, meaning that consciously thinking about synaesthesia can inferfere with the perception of concurrents, and synaesthesia must reach a particular level of intensity before it interrupts the experience of consciousness and becomes itself the subject of conscious awareness. I think that the idea that thinking about synaesthesia can interfere with the perception of synaesthesia might be related to the “verbal overshadowing” effect which has been described and debated about by researchers. In fairness I should point out that Mark C. Price speculated in the recently published (2013) Oxford Handbook of Synesthesia that synaesthesia could be subject to the verbal overshadowing effect. My own ideas were arrived upon independently from Price’s writing or work.  I base the ideas of synaesthesia being a threshold phenomenon which can also be interfered with by conscious thinking on a number of my own observations. In direct contradiction to what I had expected to find, my scores for accuracy for individual letters and numbers in The Synesthesia Battery (a scientifically-validated online test of synaesthesia) were lower for the numbers and letters that have colours that I find beautiful and which I have thought about to some degree, while my best accuracy was for the numbers and letters that have the dull and ugly colours. It seems the less I think about the concurrents the more accurately I can percieve them when they are evoked. I have also noticed that most of the types of synaesthesia that I experience I was not consciously aware of before I started to think about and examine the idea of synaesthesia. I never realised that I had complete stability in the colours I associate with months and days of the week till I tested myself. While I had a dim awareness of colour colouring my thoughts, I’d not realised that this worked like synaesthesia till I went looking for a pattern using simple testing. My fine motor movement-visual memories of scenes synaesthesia evokes concurrents that are so fleetingly and subtly experienced that they just feel like random thoughts, and indeed I now believe it is possible that the random thoughts of many or even all people are in fact synaesthesia of various types. I have also observed that there are some very unsubtle and intrusive types of syn that I experience, and they are typically rarely experienced and are associated with people, emotions, faces, singing voices or music that I find striking or novel as inducers. Because of the circumstances of these examples of synaesthesia, I think some kind of threshold is being breached when these types of synaesthesia are experienced by me.

The idea that one of the established defining criteria for synaesthesia, that it gives rise to perceptions or concurrents which are “consistent and generic (i.e., simple rather than pictorial)”, is wrong, and specific categories of memories of complex visual images such as faces and scenes, which are processed in the fusiform gyrus, can also be experienced as genuine synaesthesia concurrents. I base this assertion on the fact that I often involuntarily experience synesthesia concurrents of this type, and I have written about such experiences right from the first post in this blog which was published in 2010. I have also named types of synesthesia that have complex visual memories as concurrents: the strange phenomenon, fine motor task – visual place memory synaesthesia, involuntary method of loci memorization, etc. There are also many accounts or scientific observations of synaesthesia with complex visual concurrents in the scientific literature on synaesthesia.

Warning to readers

If you are thinking about copying or plagiarizing any of the text, ideas or descriptions found at this blog and presenting it as your own work without giving me (C. Wright, author of the blog “Am I a Super-recognizer?”) the proper acknowledgement and citations, then think again. If you do that you will be found out and my objection will be well publicized. If you want to make reference to this blog or any of the ideas in it, please make sure that you state in your work exactly where you first read about the ideas. If you wish to quote any text from this blog be sure to cite the post at this blog properly. There are many established citation methods. If you quote or make reference to material in this blog in your work, it would be a common courtesy to let me know about your work (I’m interested!) in a comment on any of the posts in this blog.

I started this blog with the primary intention of bringing interesting and fresh observations and ideas to the worlds of psychology, neuroscience and science in general. I want the stuff that I write about here to be discussed and noticed and cited by researchers. I enjoy noticing what appears to be my influence on the output of researchers. But I object to having my stuff plundered without acknowledgement of me and my blog. In a recent case of what I believe is plagiarism of some of my ideas, I believe the proper situation would have been for me to have been included as a third author of that paper, so fundamental was one of the allegedly plagiarized ideas to the main theme of that paper. You couldn’t expect anyone to be happy or satisfied about such a situation. You have been warned.

The “Bloody Mary Illusion” from New Scientist

http://youtu.be/cpF6CgxvvUo    Have you tried it? What did you “see”?

The Bloody Mary Illusion seems to be in some ways similar to another face perception illusion that was evoked by a video which I wrote about at this blog quite a while ago. Here’s links to some versions of the Ugly Face Illusion or the Flashed Face Distortion Effect from New Scientist:  http://youtu.be/o1gtxAIXoiY   http://youtu.be/o1gtxAIXoiY  and here’s another YouTube video not from another source:  http://youtu.be/WMTv4Cpj_8k

I wonder, do some people experience face perception distortion illusions more readily or quickly than others? Could this possibly explain why some people, including some of the people on the autistic spectrum, appear to avoid eye contact? I also wonder whether there is any relationship between face memory or face recognition ability, or facial expression reading ability and a person’s potential to experience face perception illusions. There you go academics! There’s some more ideas of mine to steal. Don’t thank me! (I’m sure the thought wouldn’t enter your heads anyway).

I’m not sure how these facial perception illusions work, but I suspect that they work on a similar principle to an auditory illusion that I’ve read about and heard, in which muted, ambiguous or perceptually confusing stimuli provoke the brain into interpreting the sounds as words. This sound illusion was apparently discovered by Diana Deutsch, a professor of psychology who studies interesting stuff like the psychology f music and perfect pitch ability. Her Phantom Words illusion can be found here: http://philomel.com/phantom_words/example_phantom_words.php

Just for fun, here’s a link to Jamie Frater’s Top 10 Incredible Sound Illusions:  http://listverse.com/2008/02/29/top-10-incredible-sound-illusions/